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Because B12 status governs homocysteine, it features in cognition research: in the randomized, placebo-controlled VITACOG trial, homocysteine-lowering B vitamins (including B12) slowed the rate of brain atrophy in older adults with mild cognitive impairment an effect concentrated in those with elevated baseline homocysteine[3]

Cell-culture research has shown that reducing NNMT was associated with less lipid accumulation in fat cells, while increasing NNMT was associated with more suggesting NNMT contributes to adipocyte function rather than simply marking metabolic dysfunction

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